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Various forms of a gene at a given locus, called alleles, expl
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Various forms of a gene at a given locus, called alleles, explain?

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Phenylketonuria (PKU) is a genetic disease caused by homozygosity for a recessive mutation in the enzyme that converts the amino acid phenylalanine to tyrosine. In the absence of this enzyme, phenylalanine and some of its derivatives accumulate in the body and cause mental retardation. If individuals are identified soon enough after birth, they can be treated by a lowphenylalanine diet for the early years of their lives. As adults, though, homozygous recessive individuals are allowed to adopt a diet with normal amounts of phenylalanine. Not long after such treatments were initiated, a troubling phenomenon was observed. A high number of children born to treated mothers were mentally retarded even though the children were heterozygous for the PKU gene. Children born of treated PKU males suffered no ill effects.Can you offer an explanation as to why genetically heterozygous children of treated PKU mothers might be prone to mental retardation?Propose a solution to reduce the likelihood of mental retardation in children of treated PKU mothers.
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In what form do plants take up most soil minerals?a. as ions dissolved in waterb. as neutral saltsc. as mineral-clay complexesd. linked to particles of organic carbone. None of the above describes how plants take up minerals.
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Please review the attached specific aims page written by a biomedical engineering researcher. All the necessary aspects for the aims page have been included. What strengths, weaknesses, and other feedback can you suggest to the researcher? SPECIFIC AIMS Neurological impairments among veterans of all ages. In particular, a blast-induced TBI (bTBI) primarily results from blast waves over-pressurizing not only the brain but also the lungs, ears, and other gas-filled structures [2]. Meanwhile, a secondary mechanism follows the blast overpressure with flying debris and fragmented weaponry, which can damage any other anatomical structure and, in turn, complicate bTBI care [2]. Although children and older adults who are exposed to blast waves - whether or not they occur in the battlefield - are the options, including the use of anticoagulant drugs to prevent blood clots from forming in the lower extremities [3]. Enoxaparin - an anticoagulant with low molecular weight - has especially shown promise in preclinical time of TBI occurrence remains incomprehensive. Consequently, there is a critical need to investigate determining the optimal dosing regimens for pediatric, adult, and geriatric populations. Without this integrative approach, the full potential of enoxaparin to treat bTBI will remain unrealized, and even the youngest and oldest veterans will continue facing gradual reductions in quality of life and healthcare affordability. Pharmacokinetic (PBPK) models to fully determine the age-dependent therapeutic benefits of enoxaparin in bTBI rat models. The central hypothesis is that tailoring different dosing regimens to pediatric, adult, and geriatric rat groups will significantly affect the pharmacokinetic concentration-time profiles of enoxaparin in tissues affected by bTBI. To justify, I have formulated this hypothesis based on existing literature concerning the role of age and neuroprotective agents in bTBI outcomes, followed by the observed preclinical benefits of enoxaparin. My rationale for the proposed research is that an improved understanding of the mechanisms by and pharmacodynamic data that can complement such recovery outcomes as decreased cerebral edema and increased blood-brain barrier permeability. Certain tissues of bTBI rat models across different age groups. To achieve this aim, I will administer dosages and frequencies over a 48-hour period. I will then measure the drug versus placebo concentration levels in the blood and specific tissues at different time points following administration. My working hypothesis for Aim 1 is that dosing regimen administration will significantly affect the measured enoxaparin concentration-time profiles in bTBI-affected tissues across pediatric, adult, and geriatric rat models. AIM 2: Develop age-adjusted PBPK models of enoxaparin to predict various concentration-time profiles based on the dosing regimen used to treat bTBI. To achieve this aim, I will define the absorption drug's pharmacokinetic and pharmacodynamic properties. Validation plans include fitting the PBPK models to the experimental data from Aim 1 and conducting a sensitivity analysis to determine parameters with a PBPK models will accurately predict the enoxaparin concentration-time profiles and thereby optimize the dosing regimens for different age groups of bTBI rat models. Upon completion of both aims, the expected outcomes will include an established causal relationship expect that validated PBPK models with age-specific parameters will become valuable tools for identifying the regimens with maximum neuroprotection against subsequent bTBIs according to age. These findings should have a lasting positive impact because they will expand the scope of therapeutic enoxaparin, thus informing.
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Question Text
Various forms of a gene at a given locus, called alleles, explain?
TopicAll topics
SubjectBiology
ClassClass 11